EM FAQ: Where Does the Cancer Begin?

Photo by Laboratory of Perinatal Medicine, Department of Obstetrics and Gynecology, Ohio State University.

There is an extensive literature on the relationship of trophoblast cells to cancer. Recently, when working with other researchers on tracing the etiology of cancer cells, we looked at the hypothesis that the mother of all cancer cells is a trophoblast cell. Working with scanning electron microscope photos of trophoblasts, a candidate frequency for trophoblast cells was found. This was followed by testing almost a dozen cancer patients, both animal and human.

The remarkable finding was that in every case the candidate frequency of the trophoblast cell could be found and this frequency was stable across individuals and was the same in men, women, and dogs. Those with an FSCAN2 should run a DIRP scan between 13333000 and 13334000 with a delta of 10. You are likely to find a peak in a cancer patient at the site of origin of a tumor.

We suspect that recurrence of a tumor is highly probable without elimination of these cell types.

Trophoblasts: On the Cause of Birth And Its Relationship to Cancer Regression

Roger Cathey, Cancer Cure Foundation

Abstract: Cancer has long been recognized to share histological and behavioral characteristics with pregnancy trophoblasts. Modern methods have proven biochemical and genetic characteristics are shared between cancer and pregnancy trophoblasts. Cellular or cytotrophoblasts are the source cells for the entire placenta and are the first differentiated tissue that forms after sperm and egg unite to form the zygote. Cellular trophoblasts are invasive, eroding, and metastasizing cells, which mediate the parasitization of the mother’s uterus with a new life form which grows concomittantly and distinctly alongside the placental process. The relationship between placenta and fetus represents a unique process in terms of tissue and organ development: an extra-somatic tissue, an organ, is developed with transient functions that forms no part of the somatic complement of tissues and organs, and whose growth and development is unrestricted by intrinsic factors, that is, factors within the trophoblasts themselves. It is proposed that factors extrinsic to the trophoblasts themselves eventually cause these trophoblasts to surrender their grasp on the uterine tissues, and the fetus itself. Furthermore, that these extrinsic factors are principally hydrolytic enzymes secreted by the maternal and fetal systems, and secondarily immunological factors. This degradation and cellular destruction of the trophoblasts that comprise the interface between placenta and uterus initiates a sequence of events which eventually culminate in the birth of the baby. It is believed that these same mechanisms may be pertinent to the regression or destruction of cancer cells as well, since there is a fundamental genetic identity between cancers of all types and from all origins and pregnancy trophoblasts. Thus the cause of birth and the control of wandering trophoblasts in pregnancy may provide insight to the control and possible rational therapeusis of cancer.

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