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Jeff Sutherland

Twice the Energy with Half the Stress

Live Longer: Reduction of Calcification in the Arteries with Frequencies

Since 1975 I have been working with some of the leading medical researchers in the world. I worked with twice Nobel Laureate Linus Pauling while a professor at the U.S. Air Force Academy. Working with physicans in the Academy hospital we were contemplating a clinical trial of Vitamin C to see if it reduced colds in cadets. Alas, the administration would not permit it in 1974. However, in 1975 I joined the faculty of the University of Colorado School of Medicine and in 1980 hooked up with Dr. Pauling when he sponsored the University of Colorado Center for Vitamins and Cancer Research (which I co-founded with another senior scientist). I had millions of dollars of grant funding from the National Cancer Center every year for over a decade which got me started on advanced technologies to eliminate disease. A few years ago National Cancer Institute leadership told me I was still on the list of only 300 scientists qualified to lead large grants for cancer research.

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Linus Pauling (February 28, 1901 – August 19, 1994) was an American chemist, biochemist, peace activist, author, educator, and husband of American human rights activist Ava Helen Pauling. He published more than 1,200 papers and books, of which about 850 dealt with scientific topics. New Scientist called him one of the 20 greatest scientists of all time, and as of 2000, he was rated the 16th most important scientist in history.

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Dr. Pauling showed me his almost complete DNA model and the data he gave Watson and Crick which he claimed enabled them to publish first and achieve a Nobel prize which Dr. Pauling thought rightfully belonged to him. He is the only human to achieve two independent Nobel prizes and was gunning for a third. He radically changed my thinking about medicine by showing me his lab and introducing me to his fellow researchers.

This started me down a path that led to frequency medicine which is now emerging into the mainstream. There are 566 peer reviewed medical research papers at PubMed.gov on Pulsed Electromagnetic Frequency research and 5167 papers published on electromagnetic field therapy. This is truly the future of medicine with FDA approved cancer clinical trials now in progress. Even more interesting is using electromagnetic frequencies to reprogram DNA, a more reliable and accurate approach to CRISPR.

In 2005, I visited another leading medical researcher, Dr. Terry Grossman, also a graduate of the University of Colorado School of Medicine. He had just published a book with Ray Kurzweil, Fantastic Voyage: Live Long Enough to Live Forever. At that time, my biological age was over 20 years younger than my calendar age, almost the lowest he had ever seen (two Japanese guys had me beat).

Dr. Grossman put me on a program to regularly scan my arteries for calcification as he felt was the leading risk of death for someone who had already eliminated some cancers with frequencies (as proven by physician biopsy). I had almost no calcification on a CT scan in 2005. However, by 2012 my calcium score was elevated:

The Coronary Artery Calcium (CAC) Screening Test was done utilizing Ultra-Fast Coaxial Tomography (UF CT Scan) that was able to image the amount of calcified atherosclerotic plaque in the coronary artery walls of your heart. The computer was able to calculate a calcium score for each region of calcified plaque in each coronary artery, and a total calcium score for the heart as a whole.

There are two types of plaque that develop in the arteries: hard or calcified plaque and soft or vulnerable plaque. Heart attack risk appears to be more closely related to soft, vulnerable plaque, but at present we do not have imaging devices able to quantify this type of plaque. The ultrafast CT scan is only able to measure the heart calcified plaque. Since there is a direct correlation between hard, calcified plaque, which your CAC test measured, and soft, vulnerable plaque, higher calcium scores are related to a higher risk of heart attack. Therefore this test offers an indirect assessment of dangerous coronary atherosclerosis. Your ultrafast CT scan of 105 indicated that you had detectable calcified plaque of 105 in your coronary arteries at the 30th percentile.

On 20 December 2016, Dr. Grossman reported: Cardiovascular- Your coronary artery score increased from 15 in 2005 to 105 in 2012, placing you in the 30th percentile at that time. Now your calcium score is 399 at the 65th percentile. Explained the creation of a biofilm that protects self-replicating crystals/nanobacteria.

A new supplement called Nanobac was available at the time and was recommended. A great independently-assessed summary of research is here: Anton Kutikin, PhD in The International Journal of NanoMedicine “The Role of CNPs in Biology & Medicine” www.ncbi.nlm.nih.gov/pmc/articles/PMC3266001

This supplement eliminated some of the nanobacteria and stirred up the rest. When they were active I was able to determine frequencies and target them for elimination. On 13 October 2017, less than a year after my calcium score was 399, I visited Brigham and Women’s state of the art cardiology center in Boston to get a CT scan. Results are below:

My calcium score was 55, a better than 86% reduction. Dr. Grossman said this had never been done before in the history of medicine and results should be followed up with further research studies.

Here is exactly what I did to achieve this result (nothing is guaranteed):

  1. Worked closely with a knowledgable physician to get appropriate lab tests and followup.
  2. Purchased NanobacTX and followed instructions at https://nanobiotechpharma.com/
  3. Used nanobacteria frequency sets at https://www.frequencyfoundation.com/product/nanobacteria/
  4. Purchased frequency application system. For people new to this field try a Spooky2 system of your choice. For those with some expertise consider the Frequency Research Foundation standard lab which is much more powerful for remote work.
  5. Selected the right nanobacteria frequency sets using kinesthiology (muscle testing or a dowsing technique) and ran them while taking the recommended dosage of NanobacTX (initially 8 capsules a day for six months, followed by 2 capsules a day for maintenance).

FatBuster Bacteria Frequencies

Stomach bug makes food yield more calories

Mice with a hefty dose of a certain gut bacteria are fatter.
by Helen Pearson, May 26, 2006
[email protected]

Scientists have identified a key microbe in our guts that helps us glean more calories from food. The discovery backs the idea that the type of microbes in our gut help to determine how much weight we gain, and that seeding the intestine with particular bugs could help fight obesity…

The researchers took mice that had been grown in a sterile environment, with no microbes in their guts, and injected them with a very common strain of human intestinal bacteria, called Bacteroides thetaiotaomicron. Some of the mice also received a dose of of M. smithii.

About 100 times more microorganisms took up residence in the colon of mice injected with both B. theta and M. smithii than in those injected with B. theta alone. This suggests that the presence of waste-removing M. smithii was somehow helping other bacteria to thrive. “There’s something cool going on,” Buck says.

When both microbes were present, B. theta boosted the activity of genes involved in breaking down and metabolizing fructans a food component common in onions, wheat and asparagus that the human gut cannot digest by itself. B. theta converted the fructans into fatty acids, some of which were taken up by the mouse gut and either used as fuel or stored as fat.

In humans, around 10% of our calories come from such microbe-manufactured fatty acids. After a few weeks, mice with both types of microbe had approximately 40% more of a particular fatty acid called acetate in their blood, and carried 15% more fat.

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Lengthen Your Telomeres – The Biggest Antiaging Breakthrough in the Last Decade

The Nobel Prize in Physiology or Medicine 2009 was awarded jointly to Elizabeth H. Blackburn, Carol W. Greider and Jack W. Szostak “for the discovery of how chromosomes are protected by telomeres and the enzyme telomerase”.
 
In October 2016 I visited Dr. Grossman’s lab to get telomere lengths tested. A telomerase program I developed was running for several weeks prior to the visit. More later when I get lab tests back.
 
In 2016, Dr. Grossman recommends a new supplement TeloSC from MaxLife Solution as the best and least expensive option that provides stem cell support as well as telomere enhancement.

When I visited Dr. Grossman’s medical clinic in 2012 extensive testing determined that my biological age was 27 years younger than my calendar age. I asked Dr. Grossman what the biggest breakthrough was in antiaging since my previous visit 6 years earlier. He said TA65, a supplement that costs $500 a bottle.Dr. Al Sears put me on to TA65 years earlier and he has been working on a supplement that costs less than half as much that works a lot better. Lengthening your telomeres could improve your health and performance and give you another 20 years on your life which is long enough to capture the next wave of longevity technology. See Kurzweil and Grossman’s book, Fantastic Voyage: Live Long Enough to Live Forever. But first, let’s look at the Genetic Learning Center at the University of Utah and learn a little bit about why telomeres are so important.
are_telomeres_the_key_to_aging_and_cancer

Fluorescence-stained chromosomes (red) on a microscope slide.
Telomeres (yellow) sit at the ends of each chromosome.
Photo courtesy of Dr. Robert Moyzis, UC Irvine, US Human Genome Program

Inside the nucleus of a cell, our genes are arranged along twisted, double-stranded molecules of DNA called chromosomes. At the ends of the chromosomes are stretches of DNA called telomeres, which protect our genetic data, make it possible for cells to divide, and hold some secrets to how we age and get cancer.
Telomeres have been compared with the plastic tips on shoelaces, because they keep chromosome ends from fraying and sticking to each other, which would destroy or scramble an organism’s genetic information.
Yet, each time a cell divides, the telomeres get shorter. When they get too short, the cell can no longer divide; it becomes inactive or “senescent” or it dies. This shortening process is associated with aging, cancer, and a higher risk of death. So telomeres also have been compared with a bomb fuse. For more see Learn Genetics …

I’ve been working with Dr. Al Sears new supplement Telo-Essence II for the past couple of months. He claims it is 100 times better than the TA65 which I have been taking for several years. What I can report is that it has a significantly bigger impact on energy level and physical performance.

For me, the number one antiaging strategy today, based on Nobel Prize level research is lengthening your telomeres and Dr. Sears has got this figured out. You can get Telo-Essence II here. You will need to scroll down when you reach the link. I continue to use this in conjunction with TeloSC.

 
 

Sugar in Soda Shortens Teomeres

 2014 Dec;104(12):2425-31. doi: 10.2105/AJPH.2014.302151. Epub 2014 Oct 16.

Soda and cell aging: associations between sugar-sweetened beverage consumption and leukocyte telomere length in healthy adults from the national health and nutrition examination surveys.

Abstract

Objectives. We tested whether leukocyte telomere length maintenance, which underlies healthy cellular aging, provides a link between sugar-sweetened beverage (SSB) consumption and the risk of cardiometabolic disease. Methods. We examined cross-sectional associations between the consumption of SSBs, diet soda, and fruit juice and telomere length in a nationally representative sample of healthy adults. The study population included 5309 US adults, aged 20 to 65 years, with no history of diabetes or cardiovascular disease, from the 1999 to 2002 National Health and Nutrition Examination Surveys. Leukocyte telomere length was assayed from DNA specimens. Diet was assessed using 24-hour dietary recalls. Associations were examined using multivariate linear regression for the outcome of log-transformed telomere length. Results. After adjustment for sociodemographic and health-related characteristics, sugar-sweetened soda consumption was associated with shorter telomeres (b = -0.010; 95% confidence interval [CI] = -0.020, -0.001; P = .04). Consumption of 100% fruit juice was marginally associated with longer telomeres (b = 0.016; 95% CI = -0.000, 0.033; P = .05). No significant associations were observed between consumption of diet sodas or noncarbonated SSBs and telomere length. Conclusions. Regular consumption of sugar-sweetened sodas might influence metabolic disease development through accelerated cell aging.

Pseudoscience: How Big Pharma Cooks the Books in the Scientific Journals

When David Sinclair’s company started clinical trials on the health benefits of reverse ageing through manipulation of genes, big pharma published critical articles on his approach and the FDA shut down his clinical trials. Fortunately, David did not capitulate to corporate manipulation of the scientific journals.

Time Magazine Recognizes Dr. David Sinclair for His Work On Nad+ and Resveratrol’s Contribution to Human Health and Disease Prevention.

Posted by  on May 8th, 2014 // No Comments
LIGHTNING RELEASES — Arlington, Virginia, Biotivia, May 8th, 2014. Dr David Sinclair fought a lonely battle for 5 years. As the Harvard scientist who discovered the health benefits of Resveratrol, a molecule found in red wine grapes, Sinclair’s work was dismissed by his critics. Now with thousands of scientific studies confirming his claims, Sinclair is getting the recognition he deserves. 
Time Magazine just added Dr Sinclair to their list of 100 Most Influential persons, and in December of last year David and a team of fellow scientists from Harvard, MIT, The National Institutes of Health and UNSW announced in the journal Nature that they have discovered that a coenzyme called NAD+ is able to reverse muscle aging by the equivalent of more than 30 human years by restoring communication between the cells’ nuclear and mitochondrial DNA.

Dr. Jonathan Wright reports:

The extent of published research on the phytochemical resveratrol is impressive and growing all the time, with now more than 6000 studies reported. This pharmacologically promiscuous compound demonstrates an amazing array  of favorable health outcomes, such as cardioprotective, antidiabetic, anticancer, antiviral, neuroprotective, antiplatelet, anti-inflammatory and modulation of fat metabolism. More importantly, the development of major chronic diseases might also be reduced by resveratrol, based on laboratory studies, including cardiovascular disease, dementia, type 2 diabetes and osteoarthritis. This is in addition to its touted effect on holding back the aging process via SIRT1, implying that this one simple molecule has the potential to help prevent most of the chronic diseases associated with old age. For more see Nutrition and Healing, Volume 21, Issue 5, August 2014.

Pseudoscience: Rampant in Modern Medicine

Much of today’s medical information is distorted by business interests. With billions of dollars at stake, anything can be distorted and information is often suppressed about cheap, effective solutions, in favor of expensive medications with marginal benefit. Resveratrol issues are only the tip of a very large iceberg.

How Modern Medicine Obfuscates Resveratrol Science

Marketing a resveratrol-based dietary supplement has given me a front-row seat to view how modern medicine obfuscates science and throws in other roadblocks to indefinitely delay public acceptance of a truly miraculous natural molecule.

Examination of events and published studies involving the red wine molecule resveratrol (rez-vair-a-trol) over the past decade reveals nine ways modern medicine has attempted to muddy the science and delay public adoption of this natural molecule as an affordable dietary supplement.
This investigation reveals that researchers (a) intentionally employ overdoses of resveratrol in laboratory studies to produce negative or null results; (b) absurdly claim resveratrol is not biologically available when systemic results have been widely reported in animals and humans; (c) rigidly assert a single-gene target (Sirtuin1) is responsible for most of the health benefits produced by resveratrol when aging and chronic disease involves many genes; (d) doggedly pursue development of synthetic resveratrol-like molecules (analogs) in a futile attempt to produce a blockbuster drug; (e) ignore evidence that resveratrol works better at lower doses (hormesis); (f) continue to ignore an available resveratrol dietary supplement that has been demonstrated to produce health benefits in the animal lab and humans greater than a resveratrol-based drug that sold to a major drug company for $720 million; (g) launch false allegations against resveratrol researchers who conduct studies involving branded resveratrol dietary supplements; (h) produce faulty science with flawed conclusions; and finally (i) simply fail to prescribe or recommend resveratrol to needy patients.
Some of the efforts to obfuscate the science surrounding resveratrol are almost laughable as one study found efforts to alter its molecular structure in order to produce a patentable blockbuster resveratrol-like drug were futile. After molecular side chains are added to resveratrol they are efficiency removed during liver metabolization and return to native resveratrol.
Another absurdity is the false assertion resveratrol is not biologically available due to its attachment to detoxification molecules as it passes through the human liver while animal and human studies document profound systemic health benefits.
Hyperlinking permits presentation of published reports and events that can be checked by readers themselves.

Dr. Eternity – Frontier Medical Institute Revisited

A few years ago I wrote about my visit to the Frontier Medical Institute (now Grossman Wellness Center) to have my biological age checked. Dr. Grossman’s comment about my work on frequency medicine is that it is beyond anything in his book, Fantastic Voyage: Live Long Enough to Live Forever. I get a physical every five years whether I need it or not and will be back there to see if I got younger sometime soon. A great article on Terry’s clinic appeared in the Denver Westwood magazine (see below).

In 2010, Ray Kurzweil and Terry Grossman published a new book, Transcend: Nine Steps to Living Well Forever. This is helpful in framing your own longevity program and strongly recommended.

For a systematic analysis of the science behind life extension, read Aubrey de Gray’s book on Ending Aging: The Rejuvenation Breakthrough. The Frequency Foundation has already developed several frequency strategies addresses his key targets for life extension.

Doctor Eternity
If Terry Grossman lives forever, he wants you to be there to see it.
By Joel Warner
Westword, June 12, 2008

My quest for eternal life begins at 8 a.m. in a place called the Grossman Wellness Center, with ten vials of blood and a bottle of what tastes like Sunny Delight.

From the outside, the sleek building in Lakewood looks nothing like a medical office. But the high-tech location makes sense: Inside, I’m being poked and prodded and wired up and calibrated and deconstructed and reconstructed like a very complicated machine.
Click here for more …

Longevity Futures: Reverse Aging

Cutting-Edge Natural Product TA-65 Turns On Longevity Gene

Leslie J. Farer
In the past, halting the aging process was the goal of longevity enthusiasts. Now, research shows that it is possible to not just stop, but to reverse some of the infirmities associated with growing older and rejuvenate age-damaged tissues. Ensuring the integrity of telomeres, small segments of repeating DNA at the ends of our chromosomes that decay with time, has the potential to greatly extend youthful vitality and vibrant health. A novel concentrated plant extract known as TA-65 acts at the genetic level to maintain the structure of DNA, restore cellular function, and possibly keep you years younger than your chronological age.

What are Telomeres?

Telomeres are strips of DNA located at the ends of chromosomes. They are composed of a few hundred or more repeats of the nucleotide sequence TTAGGG (1,2) (where T= thymine; A = adenine; G= guanine). They act as protective ‘caps’ preventing loss of DNA, and also guard against fusing, fraying and unraveling of chromosomes, (1,3,4) which can expose and damage the genetic material and cause mutations.
Since investigators in the 1930’s coined the term telomere (from the Greek telos for ‘end’ and meros for ‘part’), a massive amount of research has been undertaken to explore the structure and function of these important chromosomal caps. More than 8,000 scientific papers have been written and the 2009 Nobel Prize in physiology and medicine was awarded to three scientists for their work involving telomeres.

Hayflick’s Fifty Year-Old Observation

Back in the 1960’s, geneticist Leonard Hayflick observed dividing human cells and made an important discovery that had a substantial impact on the future of anti-aging research. But before we examine his finding, let’s look very briefly at cell division, or mitosis ─ a fundamental, genetically-controlled process that enables a cell to duplicate itself, including chromosomes (through the process of DNA replication), to form two identical ‘daughter’ cells. Cell division allows an organism to develop and differentiate into an adult from multiple progressive stages beginning with the initial union of reproductive cells (the germ cells, egg and sperm), and after growth and birth, to construct and repair tissues.
Hayflick reported that human cells cultured under perfect conditions in his lab divided only a finite number of times in a progressive course leading to replicative senescence, (5) when they stop dividing altogether and become dysfunctional (the term senescence is roughly equivalent to ‘aged.’). Once cells have reached their proliferation maximum, or so-called Hayflick limit, they stop functioning properly or die through the process of apoptosis. (1,6,7)
Why do cells stop dividing when they reach their Hayflick limits? Is there a cellular mechanism that monitors and counts each cell division up until the final one? And does Hayflick’s observation have any relevance to aging?
In the early 1970’s, Russian scientist Alexey Olovnikov began to answer some of these questions ─ he made the connection between a DNA replication problem involving telomeres and the limitation on cell division reported by Hayflick.

Resveratrol: Life extension effects

When an item makes the New York Times, it becomes part of the historical record. In biological sciences, the journal Nature serves a similar purpose. The definitive article on resveratrol’s life extending effects appeared in Nature this month.

Resveratrol improves health and survival of mice on a high-calorie diet
Nature 444, 337-342 (16 November 2006) doi:10.1038/nature05354; Received 10 August 2006; Accepted 19 October 2006; Published online 1 November 2006
Joseph A. Baur, Kevin J. Pearson, Nathan L. Price, Hamish A. Jamieson, Carles Lerin, Avash Kalra, Vinayakumar V. Prabhu, Joanne S. Allard, Guillermo Lopez-Lluch, Kaitlyn Lewis, Paul J. Pistell, Suresh Poosala, Kevin G. Becker, Olivier Boss, Dana Gwinn, Mingyi Wang, Sharan Ramaswamy, Kenneth W. Fishbein, Richard G. Spencer, Edward G. Lakatta, David Le Couteur, Reuben J. Shaw, Placido Navas, Pere Puigserver, Donald K. Ingram, Rafael de Cabo and David A. Sinclair

Abstract

Resveratrol (3,5,4′-trihydroxystilbene) extends the lifespan of diverse species including Saccharomyces cerevisiae, Caenorhabditis elegans and Drosophila melanogaster. In these organisms, lifespan extension is dependent on Sir2, a conserved deacetylase proposed to underlie the beneficial effects of caloric restriction. Here we show that resveratrol shifts the physiology of middle-aged mice on a high-calorie diet towards that of mice on a standard diet and significantly increases their survival. Resveratrol produces changes associated with longer lifespan, including increased insulin sensitivity, reduced insulin-like growth factor-1 (IGF-I) levels, increased AMP-activated protein kinase (AMPK) and peroxisome proliferator-activated receptor-gamma coactivator 1alpha (PGC-1alpha) activity, increased mitochondrial number, and improved motor function. Parametric analysis of gene set enrichment revealed that resveratrol opposed the effects of the high-calorie diet in 144 out of 153 significantly altered pathways. These data show that improving general health in mammals using small molecules is an attainable goal, and point to new approaches for treating obesity-related disorders and diseases of ageing.

Live Long Enough to Live Forever

I’m in Denver this week and thought I would check out the physician that Ray Kurzweil works with. Ray is a well known and successful serial entrepreneur in the Boston area. Terry Grossman MD is a leading expert on anti-aging and life extension therapies, and the founder and medical director of Frontier Medical Institute in Denver, Colorado. He is the co-author of a new book, Fantastic Voyage: The Science Behind Radical Life Extension. Dr. Grossman’s co-author is the world-renowned inventor and futurist Ray Kurzweil. Fantastic Voyage is the second book written by Dr. Grossman. In 2000, he wrote the well-received Baby Boomers’ Guide to Living Forever. One of the world’s leading proponents of anti-aging medicine, Dr. Grossman strongly believes that humanity stands on the verge of radical increases in longevity. Through his clinical practice in Denver, he has developed numerous protocols for measuring and modifying biological age and promoting longevity.

As medical director of Frontier Medical Institute, he devotes most of his professional time to running his nutritional medicine practice with emphasis on intravenous vitamin and nutritional therapies, as well as anti-aging medicine. He utilizes bio-identical hormone replacement therapy where indicated. In addition, Dr. Grossman is assistant professor of family practice at The University of Colorado School of Medicine.

I got my Ph.D. at the University of Colorado School of Medicine and was an Assistant Professor of Radiology, Preventive Medicine, and Biometrics there for many years. I’ll post a report on the experience after I go through two days of tests. Sort of like the food critic visiting a high class restaurant.

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