Electric Frequency Skin Patch: Antibiotic-Free Healing
Discover how the Electric Frequency Skin Patch is revolutionizing the treatment of bacterial infections. This innovative skin patch employs imperceptible electric currents to effectively control and eliminate microbes, providing a significant breakthrough in the era of growing antibiotic resistance. Here’s an overview of how this technology functions and why it’s considered a game-changer in medical treatments.
A Breakthrough in Infection Control
Developed by researchers eager to provide alternatives to antibiotics, this skin patch utilizes low-intensity electric currents to disrupt the growth and activity of harmful bacteria. This approach is particularly advantageous for treating skin infections and enhancing wound healing processes, marking a significant advancement in bioelectronic medicine.
How the Electric Frequency Skin Patch Works
The technology behind the skin patch is as fascinating as it is effective. By generating imperceptible electric currents, the patch targets microbial cells selectively, impairing their ability to grow and multiply. This mechanism of action localizes the treatment, reducing the risk of systemic side effects – a common concern with conventional drug therapies.
Clinical Applications and Promising Outcomes
The versatility of the electric frequency skin patch extends beyond basic infection control. Notably, it addresses chronic wounds, surgical infections, and other skin-related microbial issues. Moreover, recent studies have validated the patch’s efficacy, underscoring its potential as a viable alternative to antibiotics, particularly in scenarios where drug resistance is a significant challenge.
Published Findings and Future Potential
The Cell Press journal, Device, published the exciting results of this research on October 24, 2024. University of Chicago’s Bozhi Tian leads the study, confirming the patch’s effectiveness and potential in disease management. Explore the complete study details in ‘Bioelectronic drug-free control of opportunistic pathogens through selective excitability’. Check out our podcast on this disruptive innovation.
Transforming Healthcare with Frequency Technology
At the Frequency Research Foundation, we recognize the immense potential of frequency-based technologies like the electric frequency skin patch. By integrating such innovations into our therapeutic arsenal, we aim to enhance patient outcomes while minimizing reliance on pharmaceuticals. This aligns with our broader mission to pioneer advanced, non-invasive treatments that harness the power of frequencies to heal and protect the human body.
Explore Cutting-Edge Treatments
Interested in how frequency technology can revolutionize your health care strategy? Book a meeting to explore our innovations. Learn how we’re leading with frequency-based health solutions.
Combatting Whooping Cough
Whooping cough, or pertussis, continues to pose a significant health risk, particularly to infants and young children, despite available vaccinations. This blog explores the crucial aspects of it, including its symptoms, how it spreads, and effective prevention and treatment strategies.
What is Whooping Cough?
Whooping cough is a severe respiratory infection caused by the Bordetella pertussis bacterium. It’s characterized by intense coughing fits followed by a distinctive “whooping” sound during the next breath. While vaccines can prevent the disease, outbreaks still occur, making public awareness and vaccination crucial.
Dive deeper into our discussion on whooping cough by listening to our comprehensive podcast, where we explore more about prevention and the latest treatments. Listen to our podcast here.
Its Symptoms
The progression of whooping cough can be divided into three stages:
Catarrhal Stage: This initial phase resembles a common cold, with symptoms like a runny nose, sneezing, and a mild cough.
Paroxysmal Stage: After one to two weeks, severe coughing fits occur, which may end with a high-pitched whoop sound.
Convalescent Stage: The cough gradually improves but can last for months.
Who is at Risk?
The most at-risk groups include:
Infants and young children: Particularly those too young to have completed the full course of vaccinations.
Unvaccinated individuals: Including those who have not received booster shots.
Pregnant women: Who can pass the bacteria to their newborns.
Prevention Through Vaccination
Children should receive the DTaP vaccine according to CDC guidelines, and pregnant women should get the Tdap vaccine during each pregnancy to protect newborns. However, it vaccines always worked we would not have the current CDC alert about increasing whooping cough cases. The general health of the population has been decreasing due to many known factors which have not been addressed. Vaccines are only a bandaid if immune systems are depressed as shown in the CDC reports of the recent large uptick in cases of cancer and increase in speed to death with cancer.
Effective Treatment Options
Early treatment of whooping cough is critical and usually involves antibiotics to control the symptoms and prevent spread. Supportive care like hydration and resting is also important, especially in severe cases.
Free Frequency Treatment for it
As part of our commitment to public health, our Whooping Cough Frequency Set is available for free. This set uses state-of-the-art frequency therapy to safely and effectively combat pertussis. Explore our Free Frequency Set here.
Why Immediate Action is Essential
Prompt diagnosis and treatment not only alleviate symptoms but also reduce the risk of severe complications such as pneumonia, weight loss from vomiting, and even rib fractures due to intense coughing. If you suspect you or your child has whooping cough, contact a healthcare provider immediately.
Understanding the symptoms, risk factors, and treatment options for whooping cough can help prevent the spread of this disease. Vaccination remains the cornerstone of prevention, supported by prompt medical intervention when needed. Vaccination remains the cornerstone of prevention, supported by prompt medical intervention and our free Frequency Set.
Aerosol Infection by Flu Viruses
Photo by Kickstarter Michael Gilvary Sick Building Project – view here
For years Frequency Foundation has been reporting on flu virus aerosol infection. Many of these infections are caused by airborne parasites where masks can block large particles. However, for virus particles of a few microns, masks are ineffective. This came to a head in COVID when it became clear that medical scientists do not understand basic physics. See https://www.wired.com/story/the-teeny-tiny-scientific-screwup-that-helped-covid-kill/
Frequencies are mathematic data that do not lie. They are present in a building or not. Frequencies sent to the building consistently reduce infection 100% of the time. You do not need a clinical trial to show this after you do it on hundreds of buildings with 100% success (in at least reducing infection if not eliminating it).
Furthermore, if you are skeptical, it only takes a few weeks on your building to prove it either helps or does not help. The alternative is to live with the infection continuously.
Conventional flus, spread by aerosol parasites, were radically reduced during COVID with masks, despite the propaganda masks were ineffective in stopping the COVID virus. The 6 foot separation was also a myth that has been debunked in the science journals. Do your own research. There is real data out there.
Here we just want to emphasize that aerosol infection is one of the primary routes of viral infection, particularly for COVID, and buildings that are not properly ventilated with air cleaners that kill viruses become infected. There are many companies that do sick building remediation but they are expensive and your local Starbucks, Whole Foods, Home Depot, and other vendors are not using them every week. A single really sick person can infect a store that does not have air clearers and ventilation that kill viruses and other pathogens quickly.
This is why we have the “Sick Building Service” for weekly scans for $99/month. We have many individual subscribers for home that are infected as people are well aware that mold is a critical issue. When we scan buildings using the same technology we use for people we find viruses, bacteria, and many other things in addition to mold. It is so inexpensive that some of our homes use this service every month, continuously.
It is critical for health clinics and restaurants to be aware of the Sick Building Service because these are the areas that become most infected, most often, by the most people. We find the same thing in retail and wholesale stores. Clearing these buildings with frequencies not only prevents infection of customers and employees, it significantly changes the atmosphere in the store make it feel more friendly. People linger longer and buy more products as one of the largest retail furniture outlets in Hawaii discovered.
Live Longer: Reduction of Calcification in the Arteries with Frequencies
Since 1975 I have been working with some of the leading medical researchers in the world. I worked with twice Nobel Laureate Linus Pauling while a professor at the U.S. Air Force Academy. Working with physicans in the Academy hospital we were contemplating a clinical trial of Vitamin C to see if it reduced colds in cadets. Alas, the administration would not permit it in 1974. However, in 1975 I joined the faculty of the University of Colorado School of Medicine and in 1980 hooked up with Dr. Pauling when he sponsored the University of Colorado Center for Vitamins and Cancer Research (which I co-founded with another senior scientist). I had millions of dollars of grant funding from the National Cancer Center every year for over a decade which got me started on advanced technologies to eliminate disease. A few years ago National Cancer Institute leadership told me I was still on the list of only 300 scientists qualified to lead large grants for cancer research.
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Linus Pauling (February 28, 1901 – August 19, 1994) was an American chemist, biochemist, peace activist, author, educator, and husband of American human rights activist Ava Helen Pauling. He published more than 1,200 papers and books, of which about 850 dealt with scientific topics. New Scientist called him one of the 20 greatest scientists of all time, and as of 2000, he was rated the 16th most important scientist in history.
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Dr. Pauling showed me his almost complete DNA model and the data he gave Watson and Crick which he claimed enabled them to publish first and achieve a Nobel prize which Dr. Pauling thought rightfully belonged to him. He is the only human to achieve two independent Nobel prizes and was gunning for a third. He radically changed my thinking about medicine by showing me his lab and introducing me to his fellow researchers.
This started me down a path that led to frequency medicine which is now emerging into the mainstream. There are 566 peer reviewed medical research papers at PubMed.gov on Pulsed Electromagnetic Frequency research and 5167 papers published on electromagnetic field therapy. This is truly the future of medicine with FDA approved cancer clinical trials now in progress. Even more interesting is using electromagnetic frequencies to reprogram DNA, a more reliable and accurate approach to CRISPR.
In 2005, I visited another leading medical researcher, Dr. Terry Grossman, also a graduate of the University of Colorado School of Medicine. He had just published a book with Ray Kurzweil, Fantastic Voyage: Live Long Enough to Live Forever. At that time, my biological age was over 20 years younger than my calendar age, almost the lowest he had ever seen (two Japanese guys had me beat).
Dr. Grossman put me on a program to regularly scan my arteries for calcification as he felt was the leading risk of death for someone who had already eliminated some cancers with frequencies (as proven by physician biopsy). I had almost no calcification on a CT scan in 2005. However, by 2012 my calcium score was elevated:
The Coronary Artery Calcium (CAC) Screening Test was done utilizing Ultra-Fast Coaxial Tomography (UF CT Scan) that was able to image the amount of calcified atherosclerotic plaque in the coronary artery walls of your heart. The computer was able to calculate a calcium score for each region of calcified plaque in each coronary artery, and a total calcium score for the heart as a whole.
There are two types of plaque that develop in the arteries: hard or calcified plaque and soft or vulnerable plaque. Heart attack risk appears to be more closely related to soft, vulnerable plaque, but at present we do not have imaging devices able to quantify this type of plaque. The ultrafast CT scan is only able to measure the heart calcified plaque. Since there is a direct correlation between hard, calcified plaque, which your CAC test measured, and soft, vulnerable plaque, higher calcium scores are related to a higher risk of heart attack. Therefore this test offers an indirect assessment of dangerous coronary atherosclerosis. Your ultrafast CT scan of 105 indicated that you had detectable calcified plaque of 105 in your coronary arteries at the 30th percentile.
On 20 December 2016, Dr. Grossman reported: Cardiovascular- Your coronary artery score increased from 15 in 2005 to 105 in 2012, placing you in the 30th percentile at that time. Now your calcium score is 399 at the 65th percentile. Explained the creation of a biofilm that protects self-replicating crystals/nanobacteria.
A new supplement called Nanobac was available at the time and was recommended. A great independently-assessed summary of research is here: Anton Kutikin, PhD in The International Journal of NanoMedicine “The Role of CNPs in Biology & Medicine”www.ncbi.nlm.nih.gov/pmc/articles/PMC3266001
This supplement eliminated some of the nanobacteria and stirred up the rest. When they were active I was able to determine frequencies and target them for elimination. On 13 October 2017, less than a year after my calcium score was 399, I visited Brigham and Women’s state of the art cardiology center in Boston to get a CT scan. Results are below:
My calcium score was 55, a better than 86% reduction. Dr. Grossman said this had never been done before in the history of medicine and results should be followed up with further research studies.
Here is exactly what I did to achieve this result (nothing is guaranteed):
Worked closely with a knowledgable physician to get appropriate lab tests and followup.
Purchased frequency application system. For people new to this field try a Spooky2 system of your choice. For those with some expertise consider the Frequency Research Foundation standard lab which is much more powerful for remote work.
Selected the right nanobacteria frequency sets using kinesthiology (muscle testing or a dowsing technique) and ran them while taking the recommended dosage of NanobacTX (initially 8 capsules a day for six months, followed by 2 capsules a day for maintenance).
Viruses Are Constantly Floating Down Through the Atmosphere
Deposition rates of viruses and bacteria above the atmospheric boundary layer
Aerosolization of soil-dust and organic aggregates in sea spray facilitates the long-range transport of bacteria, and likely viruses across the free atmosphere. Although long-distance transport occurs, there are many uncertainties associated with their deposition rates. Here, we demonstrate that even in pristine environments, above the atmospheric boundary layer, the downward flux of viruses ranged from 0.26 × 109 to >7 × 109 m−2 per day. These deposition rates were 9–461 times greater than the rates for bacteria, which ranged from 0.3 × 107 to >8 × 107 m−2per day. The highest relative deposition rates for viruses were associated with atmospheric transport from marine rather than terrestrial sources. Deposition rates of bacteria were significantly higher during rain events and Saharan dust intrusions, whereas, rainfall did not significantly influence virus deposition. Virus deposition rates were positively correlated with organic aerosols <0.7 μm, whereas, bacteria were primarily associated with organic aerosols >0.7 μm, implying that viruses could have longer residence times in the atmosphere and, consequently, will be dispersed further. These results provide an explanation for enigmatic observations that viruses with very high genetic identity can be found in very distant and different environments.
FatBuster Bacteria Frequencies
Stomach bug makes food yield more calories
Mice with a hefty dose of a certain gut bacteria are fatter.
by Helen Pearson, May 26, 2006 [email protected]
Scientists have identified a key microbe in our guts that helps us glean more calories from food. The discovery backs the idea that the type of microbes in our gut help to determine how much weight we gain, and that seeding the intestine with particular bugs could help fight obesity…
The researchers took mice that had been grown in a sterile environment, with no microbes in their guts, and injected them with a very common strain of human intestinal bacteria, called Bacteroides thetaiotaomicron. Some of the mice also received a dose of of M. smithii.
About 100 times more microorganisms took up residence in the colon of mice injected with both B. theta and M. smithii than in those injected with B. theta alone. This suggests that the presence of waste-removing M. smithii was somehow helping other bacteria to thrive. “There’s something cool going on,” Buck says.
When both microbes were present, B. theta boosted the activity of genes involved in breaking down and metabolizing fructans a food component common in onions, wheat and asparagus that the human gut cannot digest by itself. B. theta converted the fructans into fatty acids, some of which were taken up by the mouse gut and either used as fuel or stored as fat.
In humans, around 10% of our calories come from such microbe-manufactured fatty acids. After a few weeks, mice with both types of microbe had approximately 40% more of a particular fatty acid called acetate in their blood, and carried 15% more fat.
The Biofilm frequency sets include many strains of borrelia which tend to cause increased blood pressure. Heat (hot bath 119 degrees or infrared lamp) dropped blood pressure to normal. Further investigator indicated that borrelia biofilms were disabled by heat and frequency sets were developed to target these organisms. Repeated application of biofilm frequencies normalized blood pressure for short periods. Only with application of one of the antibiotics in the second paper below did blood pressure normalize for extended periods.
Lyme disease is a truly intractable puzzle. Scientists used to consider the tick-borne infection easy to conquer: patients, diagnosed by their bull’s-eye rash, could be cured with a weeks-long course of antibiotics. But in recent decades the U.S. Centers for Disease Control and Prevention has realized that up to one in five Lyme patients exhibits persistent debilitating symptoms such as fatigue and pain, known as post-treatment Lyme disease syndrome, and no one understands why. The problem is growing. The incidence of Lyme in the U.S. has increased by about 70 percent over the past decade. Today experts estimate that at least 300,000 people in the U.S. are infected every year; in areas in the Northeast, more than half of adult black-legged ticks carry the Lyme bacterial spirochete, Borrelia burgdorferi. Although the issue is far from settled, new research lends support to the controversial notion that the disease lingers because these bacteria evade antibiotics—and that timing drug treatments differently could eliminate some persistent infections…
Identification of novel activity against Borrelia burgdorferi persisters using an FDA approved drug library. Jie Feng, Ting Wang, Wanliang Shi, Shuo Zhang, David Sullivan, Paul G Auwaerter and Ying Zhang. Emerging Microbes and Infections (2014) 3, e49; doi:10.1038/emi.2014.53; published online 2 July 2014
Although antibiotic treatment for Lyme disease is effective in the majority of cases, especially during the early phase of the disease, a minority of patients suffer from post-treatment Lyme disease syndrome (PTLDS). It is unclear what mechanisms drive this problem, and although slow or ineffective killing of Borrelia burgdorferi has been suggested as an explanation, there is a lack of evidence that viable organisms are present in PTLDS. Although not a clinical surrogate, insight may be gained by examining stationary-phase in vitro Borrelia burgdorferi persisters that survive treatment with the antibiotics doxycycline and amoxicillin. To identify drug candidates that can eliminate B. burgdorferi persisters more effectively, we screened an Food and Drug Administration (FDA)-approved drug library consisting of 1524 compounds against stationary-phase B. burgdorferi by using a newly developed high throughput SYBR Green I/ propidium iodide (PI) assay. We identified 165 agents approved for use in other disease conditions that had more activity than doxycycline and amoxicillin against B. burgdorferi persisters. The top 27 drug candidates from the 165 hits were confirmed to have higher anti-persister activity than the current frontline antibiotics. Among the top 27 confirmed drug candidates from the 165 hits, daptomycin, clofazimine, carbomycin, sulfa drugs (e.g., sulfamethoxazole), and certain cephalosporins (e.g. cefoperazone) had the highest anti-persister activity. In addition, some drug candidates, such as daptomycin and clofazimine (which had the highest activity against non-growing persisters), had relatively poor activity or a high minimal inhibitory concentration (MIC) against growing B. burgdorferi. Our findings may have implications for the development of a more effective treatment for Lyme disease and for the relief of long-term symptoms that afflict some Lyme disease patients.
For the 35 percent of American adults who do daily battle with obesity, the main causes of their condition are all too familiar: an unhealthy diet, a sedentary lifestyle and perhaps some unlucky genes. In recent years, however, researchers have become increasingly convinced that important hidden players literally lurk in human bowels: billions on billions of gut microbes.
Throughout our evolutionary history, the microscopic denizens of our intestines have helped us break down tough plant fibers in exchange for the privilege of living in such a nutritious broth. Yet their roles appear to extend beyond digestion. New evidence indicates that gut bacteria alter the way we store fat, how we balance levels of glucose in the blood, and how we respond to hormones that make us feel hungry or full. The wrong mix of microbes, it seems, can help set the stage for obesity and diabetes from the moment of birth.
Fortunately, researchers are beginning to understand the differences between the wrong mix and a healthy one, as well as the specific factors that shape those differences. They hope to learn how to cultivate this inner ecosystem in ways that could prevent—and possibly treat—obesity, which doctors define as having a particular ratio of height and weight, known as the body mass index, that is greater than 30. Imagine, for example, foods, baby formulas or supplements devised to promote virtuous microbes while suppressing the harmful types. “We need to think about designing foods from the inside out,” suggests Jeffrey Gordon of Washington University in St. Louis. Keeping our gut microbes happy could be the elusive secret to weight control.
Two groups of beneficial bacteria are dominant in the human gut, the Bacteroidetes and the Firmicutes. Here we show that the relative proportion of Bacteroidetes is decreased in obese people by comparison with lean people, and that this proportion increases with weight loss on two types of low-calorie diet. Our findings indicate that obesity has a microbial component, which might have potential therapeutic implications.
Endoscopic biopsy showing granulomatous inflammation of the colon in a case of Crohn’s disease. IMAGE: WIKIPEDIA/NEPHRON
June 11, 2012
One of the biggest risk factors for liver, colon or stomach cancer is chronic inflammation of those organs, often caused by viral or bacterial infections. A new study from MIT offers the most comprehensive look yet at how such infections provoke tissues into becoming cancerous.
The study, which is appearing in the online edition of Proceedings of the National Academy of Sciences the week of June 11, tracked a variety of genetic and chemical changes in the livers and colons of mice infected with Helicobacter hepaticus, a bacterium similar to Helicobacter pylori, which causes stomach ulcers and cancer in humans.
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At the Frequency Research Foundation we have always found viruses associated with cancer and recent research on biofilms shows that they are always associated with cancer as well. NIH did DNA sequencing studies on over 600 biofilm species associated with gum disease. Over the last two years we have developed frequency sets for over 400 biofilm species and these frequencies are updated daily and available to Frequency Foundation Subscribers (see Paypal button on right).
Reduced Incidence of Prevotella and Other Fermenters in Intestinal Microflora of Autistic Children
Dae-Wook Kang equal contributor,
Jin Gyoon Park equal contributor,
Zehra Esra Ilhan,
Garrick Wallstrom,
Joshua LaBaer,
James B. Adams,
Rosa Krajmalnik-Brown mail
Abstract
High proportions of autistic children suffer from gastrointestinal (GI) disorders, implying a link between autism and abnormalities in gut microbial functions. Increasing evidence from recent high-throughput sequencing analyses indicates that disturbances in composition and diversity of gut microbiome are associated with various disease conditions. However, microbiome-level studies on autism are limited and mostly focused on pathogenic bacteria. Therefore, here we aimed to define systemic changes in gut microbiome associated with autism and autism-related GI problems. We recruited 20 neurotypical and 20 autistic children accompanied by a survey of both autistic severity and GI symptoms. By pyrosequencing the V2/V3 regions in bacterial 16S rDNA from fecal DNA samples, we compared gut microbiomes of GI symptom-free neurotypical children with those of autistic children mostly presenting GI symptoms. Unexpectedly, the presence of autistic symptoms, rather than the severity of GI symptoms, was associated with less diverse gut microbiomes. Further, rigorous statistical tests with multiple testing corrections showed significantly lower abundances of the genera Prevotella,Coprococcus, and unclassified Veillonellaceae in autistic samples. These are intriguingly versatile carbohydrate-degrading and/or fermenting bacteria, suggesting a potential influence of unusual diet patterns observed in autistic children. However, multivariate analyses showed that autism-related changes in both overall diversity and individual genus abundances were correlated with the presence of autistic symptoms but not with their diet patterns. Taken together, autism and accompanying GI symptoms were characterized by distinct and less diverse gut microbial compositions with lower levels of Prevotella, Coprococcus, and unclassified Veillonellaceae.
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