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Jeff Sutherland

Twice the Energy with Half the Stress

Carcinogens in Your Drinking Water

Take Aim Against Fluoride in Drinking Water

An Issue You Can Sink Your Teeth Into

March 4, 2014Dear Citizen for Health,As a supporter of CFH, we know you work to stay informed about the issues that affect your ability to maintain your health and wellness. We emailed you last week to draw your attention once again to the potential negative health effects of water fluoridation, and are working to bring you resources for staying informed, as well as opportunities to take action.
There are reasons to be concerned:
–“Fluoride amounts to public murder on a grand scale.” A shocking quote from Dr. Dean Burk, former head of the National Cancer Institute’s Cytochemistry Sector, co-discoverer of biotin and publisher of more than 250 scientific articles. Why was he so concerned about the link between fluoride and cancer? Watch an interview with Dr. Burk here.
–Residents of Bernalillo County, home of Albuquerque, take note: Your Water Utility Authority Board is considering a reversal of a decision from 2011 to stop adding fluoride to drinking water. The move would cost taxpayers $400,000 initially, and $100,000 per year thereafter – read more here.
–And we noted in our last email that the Environmental Protection Agency’s (EPA) own National Health and Environmental Effects Research Laboratory says that fluoride is a chemical with “substantial evidence of developmental neurotoxicity”. We think this bears mentioning again. And often.
The good news is that people all over the world are taking this fight to the grassroots, and we here in the U.S. must follow the lead set by nine states proposing anti-fluoridation bills last year. Stay tuned for more on this issue, and for how you can add your voice to the chorus calling for an end to forced fluoridation of our drinking water.

Frequency Treatment For Cancer Entering Mainstream

 2012 Jan 17;106(2):307-13. doi: 10.1038/bjc.2011.523. Epub 2011 Dec 1.

Cancer cell proliferation is inhibited by specific modulation frequencies.

Abstract

BACKGROUND:

There is clinical evidence that very low and safe levels of amplitude-modulated electromagnetic fields administered via an intrabuccal spoon-shaped probe may elicit therapeutic responses in patients with cancer. However, there is no known mechanism explaining the anti-proliferative effect of very low intensity electromagnetic fields.

METHODS:

To understand the mechanism of this novel approach, hepatocellular carcinoma (HCC) cells were exposed to 27.12 MHz radiofrequency electromagnetic fields using in vitro exposure systems designed to replicate in vivo conditions. Cancer cells were exposed to tumour-specific modulation frequencies, previously identified by biofeedback methods in patients with a diagnosis of cancer. Control modulation frequencies consisted of randomly chosen modulation frequencies within the same 100 Hz-21 kHz range as cancer-specific frequencies.

RESULTS:

The growth of HCC and breast cancer cells was significantly decreased by HCC-specific and breast cancer-specific modulation frequencies, respectively. However, the same frequencies did not affect proliferation of nonmalignant hepatocytes or breast epithelial cells. Inhibition of HCC cell proliferation was associated with downregulation of XCL2 and PLP2. Furthermore, HCC-specific modulation frequencies disrupted the mitotic spindle.

CONCLUSION:

These findings uncover a novel mechanism controlling the growth of cancer cells at specific modulation frequencies without affecting normal tissues, which may have broad implications in oncology.

Cancer is Always Associated with Viral and Bacterial Infections

How infection can lead to cancer

Anne Trafton, MIT News Office
How infection can lead to cancer

Endoscopic biopsy showing granulomatous inflammation of the colon in a case of Crohn’s disease.
IMAGE: WIKIPEDIA/NEPHRON
June 11, 2012

One of the biggest risk factors for liver, colon or stomach cancer is chronic inflammation of those organs, often caused by viral or bacterial infections. A new study from MIT 
offers the most comprehensive look yet at how such infections provoke tissues into becoming cancerous.

The study, which is appearing in the online edition of Proceedings of the National Academy of Sciences the week of June 11, tracked a variety of genetic and chemical changes in the livers and colons of mice infected with Helicobacter hepaticus, a bacterium similar to Helicobacter pylori, which causes stomach ulcers and cancer in humans.
——

At the Frequency Research Foundation we have always found viruses associated with cancer and recent research on biofilms shows that they are always associated with cancer as well. NIH did DNA sequencing studies on over 600 biofilm species associated with gum disease. Over the last two years we have developed frequency sets for over 400 biofilm species and these frequencies are updated daily and available to Frequency Foundation Subscribers (see Paypal button on right).

See also Helicobacter pylori evolution during progression from chronic atrophic gastritis to gastric cancer and its impact on gastric stem cells

Shattering Cancer Cells with Frequencies (technology is decades old)

This is decades old news but useful information for the uneducated. It shows how to kill cancer cells and other organisms with frequency devices. Dr. Bare did not invent this technology as it was invented by Royal Rife in the 1920’s.

Frequency Surgery: Star Trek Technology in Israel

For all you pseudoscientists out there who don’t believe frequencies are the future of medicine, take a look at the latest surgery technique in Israel, totally based on sound frequencies.


About 10 years ago, at the MIT Future of Health Technology annual conference I demonstrated the use of the FSCAN to identify frequencies for pathogens and then use the same frequencies for eliminating the pathogens. I was moved to do this after a COMPAQ presentation of an ultrasound device that scanned the patient and then used ultrasound frequencies to heal the patient. This was the first commercial product that is the forerunner of Star Trek Tricorder technology. You scan and heal the patient with the same frequency device.

It is easier to bring sound frequencies into the operating room because ultrasound has gone through decades of clinical trials. Electromagnetic frequencies are in earlier stages and can be see in Bill Doyle’s Ted Talk on treating brain cancer with EMF in FDA clinical trials.

Carcinogenic Properties of Roundup

 2013 Jun 8. pii: S0278-6915(13)00363-3. doi: 10.1016/j.fct.2013.05.057. [Epub ahead of print]

Glyphosate induces human breast cancer cells growth via estrogen receptors.

Source

Environmental Toxicology Program, Chulabhorn Graduate Institute, Kamphaengphet 6 Road, Laksi, Bangkok 10210, Thailand.

Abstract

Glyphosate is an active ingredient of the most widely used herbicide and it is believed to be less toxic than other pesticides. However, several recent studies showed its potential adverse health effects to humans as it may be an endocrine disruptor. This study focuses on the effects of pure glyphosate on estrogen receptors (ERs) mediated transcriptional activity and their expressions. Glyphosate exerted proliferative effects only in human hormone-dependent breast cancer, T47D cells, but not in hormoneindependent breast cancer, MDA-MB231 cells, at 10-12 to 10-6 M in estrogen withdrawal condition. The proliferative concentrations of glyphosate that induced the activation of estrogen response element (ERE) transcription activity were 5-13 fold of control in T47D-KBluc cells and this activation was inhibited by an estrogen antagonist, ICI 182780, indicating that the estrogenic activity of glyphosate was mediated via ERs. Furthermore, glyphosate also altered both ERα and βexpression. These results indicated that low and environmentally relevant concentrations of glyphosate possessed estrogenic activity. Glyphosate-based herbicides are widely used for soybean cultivation, and our results also found that there was an additive estrogenic effect between glyphosate and genistein, a phytoestrogen in soybeans. However, these additive effects of glyphosate contamination in soybeans need further animal study.
Copyright © 2013. Published by Elsevier Ltd.

PMID:

 

23756170

 

[PubMed – as supplied by publisher]

Sodium Bicarbonate and Cancer

Bicarbonate Increases Tumor pH and Inhibits Spontaneous Metastases

  1. Robert J. Gillies3

  1. 1Arizona Cancer Center, University of Arizona, Tucson, Arizona; 2Department of Pharmacology, Wayne State University, Detroit, Michigan; and 3H. Lee Moffitt Cancer Center and Research Institute, Tampa, Florida
  1. Requests for reprints:
    Robert J. Gillies, H. Lee Moffitt Cancer Center, SRB-2, 12302 Magnolia Drive, Tampa, FL 33612. Phone: 813-725-8355; Fax: 813-979-7265; E-mail: [email protected].

Abstract

The external pH of solid tumors is acidic as a consequence of increased metabolism of glucose and poor perfusion. Acid pH has been shown to stimulate tumor cell invasion and metastasis in vitro and in cells before tail vein injection in vivo. The present study investigates whether inhibition of this tumor acidity will reduce the incidence of in vivometastases. Here, we show that oral NaHCO3 selectively increased the pH of tumors and reduced the formation of spontaneous metastases in mouse models of metastatic breast cancer. This treatment regimen was shown to significantly increase the extracellular pH, but not the intracellular pH, of tumors by 31P magnetic resonance spectroscopy and the export of acid from growing tumors by fluorescence microscopy of tumors grown in window chambers. NaHCO3 therapy also reduced the rate of lymph node involvement, yet did not affect the levels of circulating tumor cells, suggesting that reduced organ metastases were not due to increased intravasation. In contrast, NaHCO3 therapy significantly reduced the formation of hepatic metastases following intrasplenic injection, suggesting that it did inhibit extravasation and colonization. In tail vein injections of alternative cancer models, bicarbonate had mixed results, inhibiting the formation of metastases from PC3M prostate cancer cells, but not those of B16 melanoma. Although the mechanism of this therapy is not known with certainty, low pH was shown to increase the release of active cathepsin B, an important matrix remodeling protease. [Cancer Res 2009;69(6):2260–8]

Pulsed Electric Fields Kill Melanoma


The publisher’s final edited version of this article is available at Biochem Biophys Res Commun
See other articles in PMC that cite the published article.
Abstract
We have discovered a new, drug-free therapy for treating solid skin tumors. Pulsed electric fields greater than 20 kV/cm with rise times of 30 ns and durations of 300 ns penetrate into the interior of tumor cells and cause tumor cell nuclei to rapidly shrink and tumor blood flow to stop. Melanomas shrink by 90% within two weeks following a cumulative field exposure time of 120 μs. A second treatment at this time can result in complete remission. This new technique provides a highly localized targeting of tumor cells with only minor effects on overlying skin. Each pulse deposits 0.2 J and 100 pulses increase the temperature of the treated region by only 3 °C, ten degrees lower than the minimum temperature for hyperthermia effects.

Carcinogenesis: Preventing it with electronic medicine

Update: This original Rife organism appears to be a necessary condition for cancer cells to survive. Eliminating this organism causes cancer cell death. There are many strains of this organism and it is very persistent.
[trx_button type=”square” style=”filled” size=”small” align=”left” link=”https://www.frequencyfoundation.com/product/original-rife-frequencies/” popup=”no” top=”inherit” bottom=”inherit” left=”inherit” right=”inherit”]Original Rife Frequencies[/trx_button]

As medical research advances, the strategy of systematically eliminating cancer cells with electromagnetic frequencies is entering the mainstream. A recent clinical trial showed clearly that low voltage application of frequencies destroys cancer cells, although their approach would need significant modification to be fully effective. Clinical trials have begun and electromagnetic frequencies have been approved by the FDA to treat certain tumor types. A remarkable video by Bill Doyle shows frequencies disrupting growth of cancer cells.

There are several steps to  dealing with cancer cells:
1. Nutritional and lifestyle factors described elsewhere are critical
2. Eliminate the Rife BX BY complex
3. Eliminate the Gregory cancer virus
4. Eliminate parasites that promote tumor growth
5. Disrupt glucose metabolism of cancer cells
6. Eliminate viruses that cause malignant cells
7. Target specific malignant cells for elimination

——
There is abundant epidemiological data from all over the world on cancer incidence for a wide variety of tumors in animal and human populations. My thesis advisor was Editor of the Journal of the National Cancer Institute in the late 1970s and early 1980s. He gave me a stack of over 300 papers from the medical journals with carcinogenesis dose reponse curves for many different species. All the curves looked different. He told me to explain why they were all different and he would give me a Ph.D. I then spent about 8 years doing supercomputer mathematical analysis of data on various types of cancer. The final data set I focused in on for publication was data from the Third National Cancer Survey which had complete coverage from many states in the U.S. and was the best available data at the time. My thesis advisor had led the survey and I had complete access to all data at the National Cancer Institute. The core model we developed is now the generally accepted model for carcinogensis. It was initially validated for colon cancer. See:
Sutherland, JV and Bailar, JC. The multihit model of carcinogenesis: etiologic implications for colon cancer. J Chronic Dis. 1984;37(6):465-80.

For most human cancers, the data clearly shows a four hit process. This can be visualized most clearly in skin tumors. An abnormal cell appears and begins dividing and creates a small patch on the skin. Within this lesion, a cell mutates, and you have a small patch growing within a patch. This happens a third time, then a fourth time. On the fourth genetic change, the cell breaks through the mechanisms that control proliferation and is malignant. It grows uncontrollably in a fifth phase, with the right promoting environment available, and can get into the bloodstream and migrate to other parts of the body causing metastases.

I have repeatedly seen the frequencies 2008 and 2127 appear in myself and others after eating certain food. I have been able to confirm that this has happened in most people who have eaten a specific meal. This is the BX and BY “virus” form of Rife’s organism. Today, this is not believed to be a virus and some think it is a mycoplasma. Recent DNA sequencing of organisms found in cancer cells showed them to be a fungus.

These organisms are clearly not a typical virus. They immediately go systemic throughout the entire body. And it originally took days of repeated treatment for long periods with the FSCAN and/or EM6+ to clear the body of them. With newer technologies they can be eliminate a lot faster.

However, more and more evidence is appearing that shows cancer cell frequencies are specific frequencies in a virus sequence. It appears that a virus disrupting the cellular machinery is one of the critical steps (but not the only step) that produces a tumor. Therefore, eliminating the relevant viruses is critical to prevent ongoing appearance of pre-malignant or malignant cells.

I have repeatedly seen non-malignant skin tumors grow quickly and in one case apparently become malignant in the presence of BX and BY. In the presence of this organism it is common to have tumors erupt in less than a day. This helps to explain an interesting aspect of my previous research in carcinogenesis. In some tumors, prostate for example, the data show that the first malignant cell appears 35 years before clinical diagnosis on the average. In a fast growing tumor like lung cancer, the first malignant cell appears about a year before clinical diagnosis. However, it is very common to see people go downhill extremely quickly after initial diagnosis. While some of this is undoubtedly due to the shock of the diagnosis and resultant depression of the immune system, it is aggrevated by the BX and BY virus. Much of conventional cancer treatment does not eliminate the virus and may even promote its growth.

So the BX and BY organisms are strong promoters of at least stage 3 (premalignant) and stage 4 (malignant) tumors and may even promote earlier stages. As indicated in the paper by Owens DM, Wei S, Smart RC. A multihit, multistage model of chemical carcinogenesis. Carcinogenesis, Vol. 20, No. 9, 1837-1844, September 1999, promotion of early phases of tumor initiation increases the cellular population at risk of mutating into a subsequent phase. This process happens so fast in the presence of BX and BY that I believe the Rife organisms are both promoters and mutagens.

The first approach to stopping proliferation of tumors must be to eradicate the BX and BY virus. Parasites also release substances that promote tumor growth so they must be eliminated as well. However, they are not as dangerous as BX and BY. I believe if everyone was monitored for BX and BY and the infection was eliminated immediately, we could significantly cut cancer incidence in the U.S., probably by more than 50%.

The fact that this information has been available for almost 100 years now shows how closed mindedness and suppression of innovation has compromised American medicine. This phenomenon has largely been driven by business interests, initially by a director of the AMA who was ultimately convicted of fraud and conspiracy for suppressing cancer therapies, and in more recent years by the pharmaceutical industry.

The tumors I have worked with (lung, cervix, skin, colon) have all had the presence of BX and BY which can be eliminated straight away. It is important to realize when targeting the BX and BY organisms with an electronic device that when you kill one strain, another strain proliferates and the frequency changes. These organisms have very stable frequencies around 2008 and 2127, however, and I have seen them deviate only by 3HZ at the most. It is important to eliminate all of them and you must have the exact frequency to 1HZ and this frequency may vary between 2003-2010 and 2125-2130. In recent years, the frequency set for BX BY has expanded to hundreds of frequencies.

Eliminating the BX and BY organism does not stop cells which have already reached stage 4 and are malignant. In fact, all malignant tumors will not show BX and BY present. They can continue to proliferate and metastasize without BX and BY. The best way to eliminate the malignant cells is to stop the ATP metabolism in these cells while leaving normal cells unaffected. This will prevent mitosis and the cancer cells will die a normal death without proliferating. This appears to be possible with frequencies in the 11,700,000 range. These frequencies also seem to stop mitosis of stage 3 cells as well. The data in the Gorgun paper shows how this is possible.

Gorgun SS. Studies on the Interaction Between Electromagnetic
Fields and Living Matter Neoplastic Cellular Culture. Frontier Perspectives 7:2:44-59, Fall 1998.

Stopping cellular mitosis requires the exact frequency. I have seen frequencies in the range 11,600,000 – 11,900,000HZ affect tumors. Different tumors of the same type (or perhaps cells in different stages) will have slightly different frequencies. Metastatic tumors will have different frequencies. They must all be eliminated systematically, one by one.

I believe that Rife was able to affect a “cure” in almost every case because his device running at 11,700,000 had enough variability and harmonics to stop mitosis in all relevant cells, even though their frequencies may have been slightly different. The curse of modern technology is that it is so precise, most people are unable to reproduce Rife’s work.

Richard Loyd had a machine some years ago that could treat directly at 11,700,000HZ. Most of us are not so lucky. I have found some success, however, in using the FSCAN to treat all the octaves of the exact frequencies in the FSCAN range from 10-3,000,000HZ. More recent FSCAN devices work in the 11-12MHZ range. Current frequency sets provided to Frequency Foundation subscribers go well over 30MHZ to target viral patterns in the bodies energy field. It will be difficult but not impossible, in my view, to stop all cellular mitosis using a Rife device that can only treat at less than 10,000HZ.

These comments represent a working hypothesis that has been successful in many cases and not as successful in rapidly growing tumors. It needs further research and new data may alter the working hypothesis. I present it so that others can take a look and see if they can produce the same results consistently.

Milkweed Cures Skin Cancer

The sap from Euphorbia peplus is effective against human nonmelanoma skin cancers

  1. J.R. Ramsay1
  2. A. Suhrbier2,3
  3. J.H. Aylward4,
  4. S. Ogbourne2
  5. S.-J. Cozzi2
  6. M.G. Poulsen1,
  7. K.C. Baumann1
  8. P. Welburn4
  9. G.L. Redlich4,
  10. P.G. Parsons2,3
Article first published online: 27 JAN 2011
DOI: 10.1111/j.1365-2133.2010.10184.x
© 2011 The Authors. BJD © 2011 British Association of Dermatologists 2011
Issue
British Journal of Dermatology

British Journal of Dermatology

Early View (Articles online in advance of print)
Additional Information(Show All)
  1. Funding sources This work was conducted with financial assistance from Peplin Biotech Pty Ltd and an Australian Commonwealth Government Industry Research and Development Board R&D START grant.
  2. Conflicts of interest J.H.A. and G.L.R. were, and P.W. and S.O. are currently, employees of Peplin Inc. (formerly Peplin Biotech Pty Ltd). J.R.R., A.S. and P.G.P. were consultants to Peplin Biotech Pty Ltd. Peplin Inc. is now wholly owned by LEO Pharma.

Summary

Background  The sap from Euphorbia peplus, commonly known as petty spurge in the U.K. or radium weed in Australia, has been used as a traditional treatment for a number of cancers.
Objective  To determine the effectiveness of Epeplus sap in a phase I/II clinical study for the topical treatment of basal cell carcinomas (BCC), squamous cell carcinomas (SCC) and intraepidermal carcinomas (IEC).
Methods  Thirty-six patients, who had refused, failed or were unsuitable for conventional treatment, were enrolled in a phase I/II clinical study. A total of 48 skin cancer lesions were treated topically with 100–300 μL of E. peplus sap once daily for 3 days.
Results  The complete clinical response rates at 1 month were 82% (= 28) for BCC, 94% (= 16) for IEC and 75% (= 4) for SCC. After a mean follow-up of 15 months these rates were 57%, 75% and 50%, respectively. For superficial lesions < 16 mm, the response rates after follow-up were 100% for IEC (= 10) and 78% for BCC (= 9).
Conclusions  The clinical responses for these relatively unfavourable lesions (43% had failed previous treatments, 35% were situated in the head and neck region and 30% were > 2 cm in diameter), are comparable with existing nonsurgical treatments. An active ingredient ofE. peplus sap has been identified as ingenol mebutate (PEP005). This clinical study affirms community experience with E. peplus sap, and supports further clinical development of PEP005 for the treatment of BCC, SCC and IEC.
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