Moms2B Avoid Fluoride
During pregnancy Moms know to protect their babies by avoiding
- smoking
- alcohol
- medications
- raw meat and seafood
- high-mercury fish
AND NOW WE SHOULD ADD
- fluoride in water, food, and drinks
Why add fluoride to the list?
In 2017, a 12-year-long government-funded study was completed. It showed significant reductions in children’s IQ when their mothers were exposed to fluoride during pregnancy. In this carefully-controlled study of Mexican mother-offspring pairs by American and Canadian researchers, mothers were receiving the same fluoride doses as mothers in the US who live in communities that add fluoride dental treatment to their water.
What should pregnant women know about this study?
The results of the 2017 study by Bashash et al. included up to 299 pregnant women and their offspring. Fluoride exposure was determined by measuring fluoride in the urine of the pregnant women because that is a very reliable measure of total fluoride exposure. The researchers found a correlation between the urine fluoride of the pregnant mothers and a loss of up to 6 IQ points in their children when the children were tested at age 4 and again between 6-12 years of age. This study was published in the journal Environmental Health Perspectives in September 2017.
Is there another mother-offspring study?
Yes, a second study by Thomas et al. was presented at a conference in March 2018. The researchers reported lower IQ scores due to prenatal fluoride exposure when the children were tested between the ages of 1 and 3.
Is this new information?
Yes. While there are another 53 published studies reporting an association of fluoride exposure with a lowering of IQ in children, and over 200 animal-fluoride studies reporting damage to the brain and reduced learning and memory ability, the surprise came with the release of these mother-offspring fluoride studies. Why? Because they have clearly demonstrated, for the first time, that pregnancy is the most critical period for exposure to fluoride. The fetus now ranks as the most vulnerable of our species to fluoride’s toxicity.
Pregnant women should know about these studies in order to take the necessary steps to protect their child’s brain.
Who funded these studies?
The two mother-offspring studies were funded by the U.S. National Institutes of Health, the U.S. Environmental Protection Agency, and the U.S. National Institute of Environmental Health Sciences.
For more info on these studies use the links
in the Select-a-Topic at the top
How is a pregnant woman exposed to fluoride?
Fluoride is added to approximately 70% of public drinking water systems across the U.S. as non-consensual dental treatment. Exposure to fluoride mainly occurs by ingestion or inhalation. The greatest exposure to fluoride for the majority of Americans comes from drinking fluoridated water and using it in food preparation to make soups, rice, coffee, tea, infant formula, etc. Fluoride also has many industrial uses, so living close to a fluoride/fluorine emitting industry is also a concern. For more information on industrial releases, see the EPA’s Toxic Release Inventory for fluorides.
What water should I drink if I am pregnant? — Find out here
Questions & Concerns? Check out the Q & A
Scientific Testing of Mercury Levels in Flu Shot
Tuesday, June 03, 2014by Mike Adams, the Health Ranger
Tags: flu shots, influenza vaccines, mercury
(NaturalNews) Mercury tests conducted on vaccines at the Natural News Forensic Food Lab have revealed a shockingly high level of toxic mercury in an influenza vaccine (flu shot) made by GlaxoSmithKline (lot #9H2GX). Tests conducted via ICP-MS document mercury in the Flulaval vaccine at a shocking 51 parts per million, or over 25,000 times higher than the maximum contaminant level of inorganic mercury in drinking water set by the EPA.(1) Here are the actual results of what we found in the influenza vaccine from GSK (lot #9H2GX):
Aluminum: 0.4 ppm
Arsenic: zero
Cadmium: zero
Lead: zero
Mercury: 51 ppm
All tests were conducted via calibrated, high-end ICP-MS instrumentation as shown in these lab videos.
http://www.naturalnews.com/045418_flu_shots_influenza_vaccines_mercury.html#ixzz33aYeWz6Y
Mercury and Autism: The Smoking Gun
Acta Neurobiol Exp (Wars). 2012;72(2):113-53.
Evidence of parallels between mercury intoxication and the brain pathology in autism.
Source
Institute of Chronic Illnesses, Inc., Silver Spring, MD, USA. [email protected]
Abstract
The purpose of this review is to examine the parallels between the effects mercury intoxication on the brain and the brain pathology found in autism spectrum disorder (ASD). This review finds evidence of many parallels between the two, including: (1) microtubule degeneration, specifically large, long-range axon degeneration with subsequent abortive axonal sprouting (short, thin axons); (2) dentritic overgrowth; (3) neuroinflammation; (4) microglial/astrocytic activation; (5) brain immune response activation; (6) elevated glial fibrillary acidic protein; (7) oxidative stress and lipid peroxidation; (8) decreased reduced glutathione levels and elevated oxidized glutathione; (9) mitochondrial dysfunction; (10) disruption in calcium homeostasis and signaling; (11) inhibition of glutamic acid decarboxylase (GAD) activity; (12) disruption of GABAergic and glutamatergic homeostasis; (13) inhibition of IGF-1 and methionine synthase activity; (14) impairment in methylation; (15) vascular endothelial cell dysfunction and pathological changes of the blood vessels; (16) decreased cerebral/cerebellar blood flow; (17) increased amyloid precursor protein; (18) loss of granule and Purkinje neurons in the cerebellum; (19) increased pro-inflammatory cytokine levels in the brain (TNF-α, IFN-γ, IL-1β, IL-8); and (20) aberrant nuclear factor kappa-light-chain-enhancer of activated B cells (NF-kappaB). This review also discusses the ability of mercury to potentiate and work synergistically with other toxins and pathogens in a way that may contribute to the brain pathology in ASD. The evidence suggests that mercury may be either causal or contributory in the brain pathology in ASD, possibly working synergistically with other toxic compounds or pathogens to produce the brain pathology observed in those diagnosed with an ASD.
Vaccine Related Research: Thimerosal Alters Brain Function
J Physiol Pharmacol. 2012 Jun;63(3):277-83.
Sex-dependent changes in cerebellar thyroid hormone-dependent gene expression following perinatal exposure to thimerosal in rats.
Source
Department of Psychiatry, Harvard Medical School/BWH, Boston, MA 02115, USA.
Abstract
Mammalian brain development is regulated by the action of thyroid hormone (TH) on target genes. We have previously shown that the perinatal exposure to thimerosal (TM, metabolized to ethylmercury) exerts neurotoxic effects on the developing cerebellum and is associated with a decrease in cerebellar D2 activity, which could result in local brain T3 deficiency. We have also begun to examine TM effect on gene expression. The objective of this study was to expand on our initial observation of altered cerebellar gene expression following perinatal TM exposure and to examine additional genes that include both TH-dependent as well as other genes critical for cerebellar development in male and female neonates exposed perinatally (G10-G15 and P5 to P10) to TM. We report here for the first time that expression of suppressor-of-white-apricot-1 (SWAP-1), a gene negatively regulated by T3, was increased in TM-exposed males (61.1% increase), but not in females; (p<0.05). Positively regulated T3-target genes, Purkinje cell protein 2 (Pcp2; p=0.07) and Forkhead box protein P4 (FoxP4; p=0.08), showed a trend towards decreased expression in TM-exposed males. The expression of deiodinase 2 (DIO2) showed a trend towards an increase in TM-exposed females, while deiodinase 3 (DIO3), transthyretin (TTR), brain derived neurotrophic factor (BDNF) and reelin (RELN) was not significantly altered in either sex. Since regulation of gene splicing is vital to neuronal proliferation and differentiation, altered expression of SWAP-1 may exert wide ranging effects on multiple genes involved in the regulation of cerebellar development. We have previously identified activation of another TH-dependent gene, outer dense fiber of sperm tails 4, in the TM exposed male pups. Together, these results also show sex-dependent differences between the toxic impacts of TM in males and females. Interestingly, the genes that were activated by TM are negatively regulated by TH, supporting our hypothesis of local brain hypothyroidism being induced by TM and suggesting a novel mechanism of action TM in the developing brain.