During the 70s and 80s I spent 15 years as a leader and participant in various group therapy practices and found them useful. However, I was impressed at the amount of work and time it could take to eliminate small things and how many things after years of therapy were not possible to eliminate.

In frequency work I do not have the time or inclination to do therapeutic work. However, often people need some so I recommend a simple tapping technique which can sometimes do in a few minutes what cannot be done in years of therapy. Everyone should learn this as a child and use it regularly as needed. You can learn this on your own using information and videos available on the web, much of it for free.

Emotional Freedom Techniques (EFT) is a system of tapping on energy meridians in the upper part of the body while thinking about a negative event. Thinking about the event generates one set of signals in the brain and tapping generates another which disrupts the first.

Symptoms that people have from negative effects are caused by the signals in the brain that constantly regenerate and become more powerful by thinking about the event. The disruptive tapping signal disconnects the thinking signal from physical symptoms. Removing the reinforcing effect of the physical symptoms allows the repetitive signaling to fade away, along with the problem. This can often happen quite quickly in a few minutes.

Social Anxiety Solutions has multiple descriptions of how it works.

Mechanoreceptors are specialized receptors that respond to mechanical forces such as tapping, massaging, or holding. Among their types: Meissner corpuscles, Pacini corpuscles, Merkel discs, and Ruffini corpuscles.

They are sensitive to stimulation on the surface of the skin anywhere on the body.
The acupuncture points (the points tapped on with EFT), called “hsue” in traditional Chinese medicine, are loci that have a particularly high concentration of mechanoreceptors, free nerve endings, and neurovascular density.

The signals that are initiated when tapping hsue travel as afferent stimuli that are capable of reaching the cortex, the amygdala, and the hippocampus.

Mechanoreceptors are distributed all over the skin surface. The signal that is generated by tapping travels via large myelinated fibers, ascends ipsilaterally through the medial lemniscus, and triggers the somato-sensory cortex at the parietal lobes and the prefrontal cortex.

From there, the signal reaches the amygdala, hippocampus, and other structures where the emotional problem has neurological entity, and the signal apparently disrupts established patterns.

After EFT treatment there are reductions in cortisol, a primary stress hormone, and these reductions are accompanied by improvements in heart rate variability (HRV).

HRV and cortisol are primary stress markers for a wide variety of genetic, hormonal, and neurological effects of stress. They both correlate with significant changes in the conditions measured on the SA-45 questionnaire (Church, 2008c).

Salmonella paratyphi b

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Today, the Hawaii Department of Health announced a Salmonella paratyphi b outbreak linked to ahi tuna.  Interestingly, the ten illnesses in Hawaii appear to be linked to 13 other illnesses nationally, and possibly even linked to an outbreak of illnesses linked to the same product two years ago.  (We reported on this phase of the outbreak in February 2008).  Was it a frozen product that was held at the manufacturer or distributor’s facilities for two years, and finally released now?  Or is there a persistent source of the paratyphi b strain at the manufacturer’s facilities?

The paratyphi b strain of salmonella is different from those that we see most frequently in foodpoisoning outbreaks.  There are well over 2,000 individual strains of salmonella, but not all of them cause typhoid fever.  Paratyphi b, however, does cause typhoid fever. 

Typhoid fever is the most serious form of enteric fever, with humans being the sole reservoir of the bacteria. Based on a recent survey, the global number of typhoid cases in 2000 exceeded 21,000,000, with more than 200,000 deaths [1]. Enteric fever, that is typhoid and paratyphoid fevers, is the common name for infections caused by Salmonella enterica serotypes typhi and paratyphi. Of the three types of S. paratyphi (A, B, and C), B is the most common.

http://casesjournal.com/content/1/1/403.  Salmonella typhi and paratyphi are also more likely than other salmonella serotypes to cause enteric fever, as well as the chronic carrier state.

Typhoid fever, or enteric fever, differs from the typical salmonella gastrointestinal illness in that typhoid fever does not always cause severe gastrointestinal symptoms. 

The syndrome of enteric fever is characterized by prolonged sustained fever, relative bradycardia, hepatosplenomegaly, rose spots, and leucopenia and neutropenia [3]. After an incubation period of 5 to 21 days (generally 7 to 14 days), fever and malaise develop, often associated with cough. A small proportion of patients may have diarrhea during the incubation period. The fever tends to rise in stepwise fashion over the first few days to a week and then becomes sustained, usually at 39.4 to 40°C (103 to 104°F) or higher. After 2 weeks of illness, the severe complications of intestinal hemorrhage or perforation may be observed. The illness usually resolves by the end of the fourth week in an untreated patient. Relapse may occur in untreated as well as treated patients, but the illness is milder than the original episode. Rarely, some of the following complications may occur: pancreatitis, cholecystitis, infective endocarditis, pneumonia, hepatic or splenic abscess, orchitis, or focal infection at virtually any site [6].

The final step in migrating with Google to a new URL for Dr. Jeff Sutherland’s Electronic Medicine is to allow direct access at http://blog.frequencyfoundation.com.

Please update your URL for this blog to http://blog.frequencyfoundation.com

The (r)evolution of cancer genetics
Francesca D Ciccarelli
Department of Experimental Oncology, European Institute of Oncology, IFOM-IEO Campus, Via Adamello 16, 20139 Milan, Italy

BMC Biology 2010, 8:74doi:10.1186/1741-7007-8-74
The electronic version of this article is the complete one and can be found online at: http://www.biomedcentral.com/1741-7007/8/74

Recent advances in sequencing technologies and the launching of massive resequencing projects such as the Cancer Genome Project [1] have boosted the production of cancer genomics data. In the past few years, the entire repertoire of human exons has been sequenced in glioblastoma [2], pancreatic [3], breast and colorectal [4] cancers, and somatic mutations in selected genes have been mapped in multiple samples of renal [5] and lung [6] adenocarcinomas. In addition, the whole genomes of individuals affected by leukemia [7,8], melanoma [9], glioma [10], breast [11,12], and lung [13] cancers have been fully resequenced. All these studies have led to the identification of more than 1,000 potential cancer genes, and the list is likely to grow in the near future.

This massive amount of information will have a huge impact on our understanding of cancer genetics, even more so considering that the biological role of most mutations is still obscure. These first unbiased screenings have led to the identification of novel and unsuspected determinants of cancer, such as the isocitrate dehydrogenase enzyme genes IDH1 and IDH2, which have been found mutated in glioblastoma multiforme [2]. They have also started to question some cornerstones of cancer biology, such as the description of cancer as a unique disease driven by the somatic modification of a few key regulators. The progressive identification of novel mutated genes is expanding the ‘cast of actors’ [14] whose mutations might be causally involved in driving cancer. Moreover, given the high heterogeneity of genes mutated in different cancer types (Figure 1), the overall ‘plot’ is becoming more intricate. The emerging picture suggests that there may be distinct genetic routes to reach the common aftermath of all tumorigenic processes, which is uncontrolled cell proliferation. For example, as many as 12 core pathways are disrupted in the majority of pancreatic cancers through multiple somatic mutations [3]. This opens up an intriguing scenario where the deregulation of key pathways for tumorigenesis represents only the final step of a more general perturbation of cellular activity. The cell is seen as an integrated system in which all processes form a tightly interconnected network more than as an ensemble of independent pathways. In this context, the effect of somatic mutations occurring in the cancer genome should be interpreted in the light of their broader impact on the system’s equilibrium…

Mercury filings are toxic and outgassing whenever they are disturbed. The mercury goes to all organ systems in your body and reduces their performance and capability. Check out this video.

Aubrey de Grey: “The first person to live to 1,000 might be 60 already”

Life expectancy is increasing in the developed world. But Cambridge University geneticist Aubrey de Grey believes it will soon extend dramatically to 1,000. Here, he explains why.
Ageing is a physical phenomenon happening to our bodies, so at some point in the future, as medicine becomes more and more powerful, we will inevitably be able to address ageing just as effectively as we address many diseases today.

I claim that we are close to that point because of the SENS (Strategies for Engineered Negligible Senescence) project to prevent and cure ageing. It is not just an idea: it’s a very detailed plan to repair all the types of molecular and cellular damage that happen to us over time.
And each method to do this is either already working in a preliminary form (in clinical trials) or is based on technologies that already exist and just need to be combined.
Click here for more …

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Reader wanting further information should read Aubrey’s book “Ending Aging: The Rejuvenation Breakthrought” as his views are consistent with the Frequency Foundation. Most of the “aging” phenomenon is a maintenance problem. Our systems become loaded up with viruses, bacteria, pollutants, and allergens like barnacles on a ship. Hospitals are like a dry dock where the ship is repaired after major problems. Unfortunately, the hospitals only patch the holes and are remarkably ineffective at removing the barnacles. In fact, you may acquire more barnacles simply by visiting the hospital and these nosocomial infections can make hospitals a deadly place to visit.

The core work of Frequency Foundation is developing frequency sets that systematically and harmlessly remove the barnacles on an ongoing basis. Routine maintainance prevents breakdown.

In a study released by the International Journal of Biological Sciences, analyzing the effects of genetically modified foods on mammalian health, researchers found that agricultural giant Monsanto’s GM corn is linked to organ damage in rats.

According to the study, which was summarized by Rady Ananda at Food Freedom, “Three varieties of Monsanto’s GM corn – Mon 863, insecticide-producing Mon 810, and Roundup® herbicide-absorbing NK 603 – were approved for consumption by US, European and several other national food safety authorities.”

Monsanto gathered its own crude statistical data after conducting a 90-day study, even though chronic problems can rarely be found after 90 days, and concluded that the corn was safe for consumption. The stamp of approval may have been premature, however.

Click here for more info …